Doctors have identified the third most important problem underlying type II diabetes

The title is an ironic euphemism for “doctors have misidentified the two most important problems underlying type II diabetes and thereby all but assured diabetic patients that no cure can be found.”

Nothing could be easier than to cure diabetes. I really believe that, but first one must identify the most important underlying problems. Thanks to doctors, we already have the third most important problem, insulin resistance. The solution to the problem of insulin resistance is the same as the solution to the first and second most important problems.

The first and most important problem that diabetics (and generally people with other degenerative diseases, including autoimmune diseases and cancer) have is the running of “degenerative metabolism.” The metabolism is “degenerative” because mitochondria become “impaired” in Otto Warburg’s term, or they “power down” or “degenerate” under this metabolic regime of relentless of using primarily sugar fermentation to regenerate ATP. Look at electron micrographs of mitochondria from cancer cells. They do not look anything like the pictures of healthy mitochondria in the Biochemistry textbooks.

This sad fact of running degenerative metabolism is yelling at doctors to stop feeding diabetics carbohydrates (the American Dietetic Association recommends a whopping 300 grams of carbohydrate a day) – doctors are forcing type II diabetics to keep running this degenerative metabolism, because carbohydrates are digested to sugars, sugars are the only fuel we can ferment, and we can completely oxidize sugars to carbon dioxide and water, only if one enzyme is working properly, mitochondrial pyruvate dehydrogenase, an enzyme requiring potassium and magnesium, which many diabetics tend to be deficient in, and most importantly an enzyme requiring thiamine, a vitamin that diabetics are urinating out of their bodies because they do not reabsorb it properly from their glomerular filtrates, and their kidneys are under serious diuretic pressure from their high sugar, and often from prescribed diuretics (for their hypertension) and caffeine.

The solution to degenerative metabolism is to jump start mitochondrial respiration with proper nutrition, sugar restriction, proper supplementation, proper fasting, and proper exercise.

The second most important problem facing the type II diabetic is lack of sugar demand by their muscles – the solutions to this problem are to create sugar demand each and every day by daily fasting, at least 18 hours, plus as vigorous an exercise program as their physical fitness allows in at least the last hour before breaking the fast, and preferably similarly vigorous exercise now an then during the long fast to keep their metabolic rate suitably high (otherwise as diabetics fast and do not exercise, their thyroids lower their metabolic rate and this reduces the rate at which they can lose weight).

Even without fasting, just by exercising, an important glucose transporter (GLUT4) moves to the cell membrane from inside the cell (the endoplasmic reticulum [ER] membranes, where it relocates to, and where it resides, when sugar demand is low. This relocation of GLUT4 transporters to the ER makes the muscle cell without a serious glucose demand look a tad more “insulin resistant” to the eye of a doctor who mistakenly thinks insulin resistance is the main problem), making the cell better able to absorb glucose from the bloodstream, and thus reducing the need for insulin, and just as importantly reducing the rate at which insulin is produced (a rate which is often too high in the diabetic who consumes too much sugar, forcing too fast a processing of pre-pro-amylin, an amylin precursor protein, a protein with clear solubility problems (even the processed protein amylin is not very soluble), which leads inevitably to precipitation and plumbing problems, problems which are aggravated by a lack of daily fasting, which would have cleared out much of the rubbish by autophagy.


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